As it turned out, in people with an accumulation of beta-amyloids in the hippocampus (this is considered a hallmark of Alzheimer's disease) as a result of regular aerobic training during the year, the development of the disease slows down.
Prof Zhang is affiliated with the department of neurology, neurotherapeutics and internal medicine at the University Of Texas Southwestern Medical Center in Dallas.
As you know, in people with a low level of physical fitness, the volume of vital nerve fibers in the brain called white matter decreases faster than in physically active ones.
However, "if these findings can be replicated in a larger trial, then maybe one day doctors will be telling high-risk patients to start an exercise plan", he said. In fact there is no harm in doing so now. These subjects were randomized into different activity groups for 12 months, including things like aerobic exercise or stretching and muscle toning.
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However, Trump and Pompeo were quick to point the finger at Tehran, and the secretary of state called the attack an "act of war" . In an interview with CNN , Zarif again denied any involvement, calling Saudi evidence "lies and deception".
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All of the patients had some amyloid plaque buildup in their brains at the beginning of the study, and were classed as having "mild cognitive impairment", often a precursor to Alzheimer's. However, brain imaging revealed that the participants with amyloid from flexibility training group experienced less reduction in the hippocampus volume. But the research suggests exercise could help. "Although the interventions didn't stop the hippocampus from getting smaller, even slowing down the rate of atrophy through exercise could be an exciting revelation".
In this week's edition of the round-up: obesity is associated with abnormal bowel diets, independent of diet and lifestyle; weekly exercise may slow the progression of Alzheimer's disease; many adults do not place a high importance on diabetes treatment guidelines; and there exists a link between childhood behavioral problems and adulthood insomnia.
Harrison will work to create a better understanding of specific biochemical pathways involved in the formation of brain lesions, a major component of the disease, by looking at how these specific pathways can be modified to promote cell survival and maintain the health of cells that are typically damaged by Alzheimer's disease. Rong Zhang. The group members launched their results in the Journal of Alzheimer's Disease.